Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. However, our capability to rescue SERT functionality in KI midbrain synaptoneurosomes through the inhibition of FAK indicates elevated FAK signaling downstream on the Pro32Pro33 mutant, as verified by amplified pFAK localization in 5-HT synapses. Our https://johnz086coz8.activosblog.com/profile